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www.aasraw.com losing weight, 14 lbs in 4 weeks. That time span is indicative of permanent loss. My wife has lost 9 lbs, and she suffers from a rare disorder called Erythromelalgia, which causes the extremities to get very hot with neuropathic pain, and it is consistent. The Niacin part of the compound, in addition to her medicines, has made the pain lessen somewhat, and enabled more functionality. After I ran out the first time – I tried the one from another NMN powder suppliers. It’s compound is different, and the energy dissipated. That’s why I came back to MAAC10, and now I’m back on track. You know, I even believe some of my widespread gray hair has darkened without dye. It works You know – I don’t think I look 57 anymore either.Anyway,thank for this miracle supplements-NMN powder and thank for aasraw.

While the Fountain of Youth remains a fantastical myth that isn’t likely to come true any time soon, the science behind anti-aging medication continues to advance. Perhaps one day, you’ll be able to take a miracle pill and experience life as a young adult once again. Studies in animals showed NMN’s promising properties in NAD+-boosting and anti-aging. Now, Although the researchers are moving forward with clinical trials to investigate the safety and efficacy of the molecule in humans.Until then, NAD and NMN supplements offer real benefits to slow the drastic effects of cell aging on the human body. I believe that it will be helpful as long as they are benefical for our body, Let’s get young again together.

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Reference

[1] North, B. J., Rosenberg, M. A., Jeganathan, K. B., Hafner, A. V., Michan, S., Dai, J., … & van Deursen, J. M. (2014). SIRT2 induces the checkpoint kinase BubR1 to increase lifespan. The EMBO journal, e201386907. [2] Kiss, T., Nyúl-Tóth, Á., Balasubramanian, P., Tarantini, S., Ahire, C., Yabluchanskiy, A., … & Ungvari, Z. (2020). Nicotinamide mononucleotide (NMN) supplementation promotes neurovascular rejuvenation in aged mice: transcriptional footprint of SIRT1 activation,

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www.aasraw.com mitochondrial protection, anti-inflammatory, and anti-apoptotic effects. GeroScience, 1-20.

[3] Grozio, A., Mills, K. F., Yoshino, J., Bruzzone, S., Sociali, G., Tokizane, K., … & Imai, S. I. (2019). Slc12a8 is a nicotinamide mononucleotide transporter. Nature metabolism, 1(1), 47-57. [4] Li, J., Bonkowski, M. S., Moniot, S., Zhang, D., Hubbard, B. P., Ling, A. J., … & Sinclair, D. A. (2017). A conserved NAD+ binding pocket that regulates protein-protein interactions during aging. Science, 355(6331), 1312-1317. [5] Stipp D. Beyond Resveratrol: The Anti-Aging NAD Fad. Scientific American Blog Network. March 11, 2015. [6] Tarragó MG, Chini CC, Kanamori KS, Warner GM, Caride A, de Oliveira GC. + Decline. Cell Metabolism. May 2018, 27 (5): 1081–1095.e10. PMC 5935140. PMID 29719225. doi:10.1016/j.cmet.2018.03.016. [7] Bogan KL, Brenner C. Nicotinic acid, nicotinamide, and nicotinamide riboside: a molecular evaluation of NAD+ precursor vitamins in human nutrition. Annual Review of Nutrition. 2008, 28: 115–30. PMID 18429699.

[8] Fletcher RS, Lavery GG (October 2018).

“The emergence of the nicotinamide riboside kinases in the regulation of NAD+ metabolism” . Journal of Molecular Endocrinology. 61 (3): R107–R121. doi:10.1530/JME-18-0085. PMC 6145238. PMID 30307159.

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