Galactosialidosis Feature

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Galactosialidosis

AnintroductiontoGalactosialidosis,anultra rareGlycoproteinDisorder

Galactosialidosisisarareglycoproteinstoragediseasecausedbyachangeinthe CTSAgene.

Butwhatdoesthismean?

TheCTSAgeneprovidesinstructionstoproduceanenzymecalled cathepsinAinourbodies.Workingalongsidetwootherenzymes, cathepsinAbreaksdowncertainsugars,proteins,andfatswithin lysosomes.Lysosomeshavearangeofimportantfunctionsfrom breakingdownworn-outcellstohelpingprotectourbodiesfrom virusesandbacteria.

ThechangetotheCTSAgenemeanssomeonewithgalactosialidosisdoesn’t produceenoughcathepsinA,whichleadstoabuild-upofcertainsubstanceswithin thelysosomesincludingglycoproteinsandglycolipids.Inturn,thisaccumulation canaffectmultipleorgansandtissuesinthebody,resultinginawiderangeof symptomsacrossthreedifferentformsofgalactosialidosis.

EarlyInfantile

Thosewiththeearly infantileformareusually diagnosedbetweenbirth and3monthsold.Infants withthisformusuallylive intolateinfancy.

Symptomsmayinclude: extensiveswelling,asoft outpouchinginthelower abdomen,anenlarged liverorspleen,abnormal bonedevelopment, enlargedheart,kidney disease,eyeabnormality, distinctivefacialfeatures.

Lateinfantile

Thosewithlateinfantile formusuallydevelop symptomswithinthefirst yearoftheirlife.Life expectancycanvary dependingontheseverity ofsymptoms

Symptomsmayinclude: Shortstature,heartvalve problems,hearingloss, eyeabnormality, intellectualdisability, distinctivefacialfeatures.

Juvenile/Adult

Theageatwhich symptomsofthisform begintodevelopcan vary,buttheaverageis16 yearsold.Thosewiththis formusuallyhavea normallifeexpectancy.

Symptomsmayinclude: Ataxiaandmuscle twitches,seizures,vision andhearingloss,eye abnormality, abnormalitiesinthe bonesofthespine,dark redspotsontheskin, progressiveintellectual disability,distinctive facialfeatures.

Galactosialidosis

AnintroductiontoGalactosialidosis,anultra rareGlycoproteinDisorder

TreatmentandMangement

Galactosialidosisisdiagnosedthroughacombinationofclinicalevaluation,genetic testing,andenzymeactivityassayswhichmeasuretheactivityofyourenzymes.The prevalenceiscurrentlyunknownbutmorethan100instancesofgalactosialidosis have beenreportedsofar.Anestimated60%ofpeoplewiththeconditionhavethe juvenile/adultform;mostofwhichareofJapanesedescent.

Thereisnotreatmentforgalactosialidosisatthemoment.Instead,careis focusedontreatingthespecificsymptomsoftheindividual,suchas takingmedicationstocontrolseizures,tohelpimprovequalityoflife.

Lifeexpectancycurrentlyvariesbetween,andwithin,thethreeforms.

TheGeneticsofGalactosialidosis

Galactosialidosisisaninheritedautosomal recessivecondition.

ThismeansthechangedCTSAgenemustbe inheritedfrombothparentsforthechildtobe affectedbygalactosialidosis.

Ifbothparentsarecarriersof galactosialidosis(andsohaveonechanged CTSAgenethemselves)there’s:

50%chancethechildwillalsobeacarrier, 25%theywillhavetwoworkinggenesand beunaffected,and 25%ofdevelopinggalactosialidosis.

ThisinformationiseditedfromtheTEMPLEGuideswhichhavebeenadaptedbytheDietitiansGroupoftheBritishInheritedMetabolic DiseaseGroup(BIMDG)andisbasedontheoriginalTEMPLEwrittenbyBurgardandWendel TheTEMPLEGuidesaresupportedbyNutricia

Galactosialidosis Network

Whilstthereisnocurrenttreatment,TheGalactosialidosisNetworkare

Clara’s Story

Clara’slifewithgalactosialidosis,herlegacy, andherfamily’scommitment

ÇağdaşandCinarCanbolatwelcomed theirbeautifulbabygirlClaraLorinonthe 30thofJune2021.

At17monthsold,Clarawasdiagnosedwith theultra-rareinheritedmetaboliccondition, galactosialidosis.

Followingherdiagnosis,Clarabattledchronic kidneydiseaseandnarrowlysurvivedaheart attackwhereherheartstoppedforeleven minutes.Sherecoveredwellbutbecame reliantondialysisfourormoredaysaweek whilstsheawaitedakidneytransplant.

Clarawasn’ttheonlyonefighting.Herfamily turnedtheirimmenseeffortsandcaretowards supportingtheirdaughterandotherswiththe ultra-rarecondition.Herfatherjoinedthe boardofISMRD(theInternationalSocietyfor Mannosidosis&RelatedDisease)toensure internationalcollaborationandtheyfounded theGalactosialidosisNetwork;bringing togetherthoseaffectedbythecondition, compilinginformationandresearch,and lobbyingforchange,development,anda spotlightontheconditionthatchangedtheir lives.

ClarapassedawayonMarch6th,2024,atjust twoandahalfyearsold.Shewillliveoninher family’smemoryandtheircontinuedsupport forthegalactosialidosiscommunity.We,too, arehonouredtobeapartofClara’smemory.

ThankyoutoClara’sfamily,friends,and communityfortheirdonationstousin Clara’snameandmemory.Togetherthey raisedover£24,500(inclusiveofgiftaid).

Meet the ISMRD

MeetTheInternationalAdvocateforGlycoprotein StorageDiseasesinthisarticlefromtheirpresident

Doyouorafamilymemberhaveoneofthefollowing?

Alpha-Mannosidosis Aspartylglucosaminuria(AGU)

Beta-Mannosidosis

Fucosidosis

Galactosialidosis

Sialidosis

SchindlerDisease

Pleasecontactusatinfo@ismrd.org.Weoffersupport,informationandconnectionwithother similarlyaffectedfamilies.Wealsoworkforresearchandtreatmentsforeachofthesediseases.

TheInternationalSocietyforMannosidosisandRelatedDiseases(ISMRD)istheInternational AdvocateforGlycoproteinStorageDiseasesgovernedbyaBoardofDirectorswhoarefamily membersfromaroundtheworldandaProfessionalAdvisoryBoardwhoaremembersofthe internationalscientificandmedicalcommunity.ISMRDhasbeenoperatingfor25yearsand membershipisfree.

OurMission

Throughpartnershipsbuiltwithmedicine, scienceandindustry,weseektodetect andcurethesediseases,andtoprovidea globalnetworkofsupportandinformation.

CurrentActivities

OurVision

Weseekafutureinwhichchildrenwith GlycoproteinStorageDiseasescanbe detectedearly,treatedeffectively,andgo ontolivelong,healthyandproductivelives.

ISMRDisassistingwiththedisseminationofLamzede,anon-centralnervoussystemenzyme replacementtherapyforAlpha-Mannosidosisadultsandchildren,thatwasapprovedforuse intheUSin2023.WearealsoworkingontreatmentsforBeta-Mannosidosis,Fucosidosisand Galactosialidosis,andfundingresearchintoAlpha-MannosidosisandMucolipidosis.

ISMRDhasprovidedpatienttestimonythathasresultedinapprovalforuseofpreimplantation genetictestingformonogenicdisorders(PGT-M)intheUK,forbothSialidosisandFucosidosis. ThismeansthatSialidosisandFucosidosisfamiliesintheUKcanhaveadditionalchildren withoutfearofthembeingaffected.Wehaveheldsixinternational conferencesandwillbeholdingour7thInternationalFamilyand ScientificConference7-10August2025inMinneapolis,Minnesota,USA. Withwarmregards, CarolynPaisley-Dew, ISMRDPresident

Youarealwayswelcometoreachouttotheteam atMetabolicSupportUK,inawaythatsuitsyou.You cancallourfreephonehelplineMonday-Friday,10am-4pm, fillinthecontactformonourwebsite,orsendusanemail.

08006523181

www.metabolicsupportuk.org

contact@metabolicsupportuk.org

@MetabolicSupportUK @weareMSUK

@MetabolicSupportUK

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