Pain Medications and Mechanisms of Action

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Pain Medications and Mechanism of Action Thomas B. Gregory, PharmD, BCPS, DASPE, CPE


Disclosure

Nothing to disclose


Objectives List pain medications and mechanism of action according to their specific therapeutic class or family Differentiate multimodal analgesia from multiple analgesic agents alone Choose medication combinations that would be effective in multimodal management of pain


httpwww.google.comimgreshl=en&biw=1400&bih=917&tbm=isch&tbnid=8AYyu6Dd2Wy6DM&imgrefurl=httpwww.changepain-emodules.comindex%3F modulesId%3D9%26languagesId%3D1&docid=YjqjkcwIlu-aTM&imgurl=httpwww.changepain-emodules.comimgm9_4.jpg&w=.jpg. Accessed May 12, 2012


Analgesic Families Opioids –Modulation of the mu opioid receptor, altering nociceptive stimuli in the central nervous system (CNS)

Non-steroidal anti-inflammatory agents (NSAID) –Inhibition of prostaglandin synthesis, preventing part of the inflammatory response

http://journals.lww.com/ejanaesthesiology/Fulltext/1997/05001/Mechanisms_of_actions_of_opioids_and_non_steroidal.3.aspx#. Accessed on August 1, 2012


Analgesic Families (cont’d)  Local Anesthetics3 – Blocking voltage-gated sodium channels in neurons

 Tricyclic Antidepressants (TCA) and Serotonin-Norepinephrine Reuptake Inhibitors (SNRI)4 – Inhibition of serotonin and norepinephrine enhances the descending inhibitory pathway in the CNS

 Antiepileptic agents5 – Inhibition of sodium channels in afferent and transmission neurons in the CNS CNS; central nervous system 3. Br Journal Anaesth.89; 1: 52-61 2002 4. Current Neuropharmacology, 7 4 331-6 2009 5. Drugs 60 5 1029-52, 2000


Analgesic Families (cont’d) Glucocorticoids6

–Alteration of intracellular gene expression, inhibiting phospholipase activity and inhibition of cytokine expression

Miscellaneous –Acetaminophen7

• Exact mechanism is unknown however thought to interact with cyclooxygenase and nitric oxide pathways in the CNS

–Capsacian8

• TRPV1 agonist which leads to defunctionalization of nociceptors CNS; central nervous system 6. http://www.practicalpainmanagement.com/corticosteroid-use-pain-management. Accessed August 1, 2012 7. Pain Physician 12; 269-80 2009 8. Br Journal of Anaesth 107; 4: 490-502 2011


Rational Pharmacology  Use of a single agent at the lowest effective dose  Rotating to another agent in the same medication family can have a positive or negative impact depending on the reason for the rotation  Other disease states often require multiple agents for maximum benefit too – Hypertension – Diabetes – Chronic pain


9 Multimodal Analgesia

Adjuvant agents in addition to opioids can lead to the same degree of pain management with lower doses of opioids Opioids are not always effective with certain painful conditions –Chronic or neuropathic pain

The use of non-pharmacologic therapy along with pharmacologic should always be considered 9. Current Opinion in Anaesthesiology 23; 6: 697–703, 2010


Rational Polypharmacy  Using a single medication from multiple families to maximize efficacy and minimize side effects  Using pain medications in multiple families to have synergistic effects – Each medication should have activity on different receptors within the pathway

 NOT using multiple agents in the same family for additive effect – Increased risk of development of side effects


10 Opiate Sparing Effects

Combination of NSAID with opioids for severe pain due to trauma Combination of TCA or SNRI with opioids for chronic pain Combination of antiepileptics with opioids for neuropathic pain Steroids as another adjuvant for acute inflammation NSAID- Non-steroidial anti-inflammatory agents TCA- Tricyclic Antidepressants SNRI- Serotonin-Norepinephrine Reuptake Inhibitors 10. Anesth Analg 101: S5–S22 2005


Short Acting Versus Long Acting Short acting opioids for severe acute pain Short acting and long acting opioids for prolonged severe acute pain or chronic pain Rapid onset opioids with short and long acting opioids for break though pain


Summary Use of rational polypharmacy can maximize efficacy in pain management and decrease the incidence of adverse events Each medication family has its advantages and disadvantages regarding specific pain conditions Formulation of the medication has just as much to do with selection as the family the medication is in does


References 1. http:www.google.comimgreshl=en&biw=1400&bih=917&tbm=isch&tbnid=8AYyu6Dd2Wy6DM&imgrefurl=http www.changepain-emodules.comindex%3FmodulesId%3D9%26languagesId%3D1&docid=YjqjkcwIluaTM&imgurl=httpwww.changepain-emodules.comimgm9_4.jpg&w=.jpg. Accessed May 12, 2012. 2. http://journals.lww.com/ejanaesthesiology/Fulltext/1997/05001/Mechanisms_of_actions_of_opioids_and_non _steroidal.3.aspx#. Accessed on August 1, 2012. 3. Br Journal Anaesth.89; 1: 52-61 2002 4. Current Neuropharmacology, 7.4. 331-6 2009 5. Drugs 60 5 1029-52, 2000 6. http://www.practicalpainmanagement.com/corticosteroid-use-pain-management. Accessed August 1, 2012 7. Pain Physician 12; 269-80 2009 8. Br Journal of Anaesth 107; 4: 490-502 2011 9. Current Opinion in Anaesthesiology 23; 6: 697–703, 2010 10. Anesth Analg 101: S5–S22 2005


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