Development of a diagnostic tool for the detection of Erwinia amylovora

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Diagnostics and genomic diversity of Erwinia amylovora Rachel Mann Research Scientist DPI VIC biosecurity built on science Cooperative Research Centre for National Plant Biosecurity


“It is said that if you know your enemies and know yourself, you will not be imperiled in a hundred battles; if you do not know your enemies but do know yourself, you will win one and lose one; if you do not know your enemies nor yourself, you will be imperiled in every single battle.”

The Art of War Sun Tzu – 600BC

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The Enemy

Australian apple orchards predominantly have root stocks susceptible to E. amylovora biosecurity built on science


Know yourself  Do we have E. amylovora or closely related species?  We know we don’t want E. amylovora  We know we have susceptible hosts  We know it is an expensive and difficult (sometimes impossible) feat to eradicate a pathogen – 1997 E. amylovora incursion $20 million  Our best line of defence is to not let E. amylovora in border detection and diagnostics

Distribution of plants in the family Rosaceae Source: Atlas of Living Australia 2012 http://biocache.ala.org.au

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Know yourself – Do we have E. amylovora?

Surveillance for E. amylovora  In 2008 we sampled E. amylovora host plants across 104 sites  200 strains of bacteria were isolated -

Selective plating ambiguous Biochemical assays PCR 16S rDNA sequencing HR Pathogenicity assays on immature pear fruit

No E. amylovora identified

112 25 72 23050A 37 14 23048B E. billingiae Eb661 39 8 AG 16 2226A 38 9 BE66 38 14 94 49 21 E. persicina Cp2 99 10 E. rhapontici Nk4-18 BE56 DAR 61733 91 BE57 BE65 E. tasmaniensis Et1/99 E. amylovora 273 DAR 75964 95 73 E. pyrifoliae Ep16/96 83 23064A P. ananatis M471 49 13 94 P. agglomerans CE21 23091B 23093A Pantoea sp. EM486 23069A 23095D 99 23046A 23085C 23086A BE62 BE68 Enterobacteriaceae bacterium BFo-2 BE55 Pear ooze A S. proteamaculans PW172 94 11 6 Rahnella sp. Pv5 R. aquatilis KNOUC601 11 22 91 11 21 11 24

82

100

99

99 89

97

75

139 24 143 10 BE63 88 P. poae BCHCNZ318 138 19 23075A P. graminis PDD-32b-66 78 100 BE3 23063A AG 16 2226B 96 P. putida strain jvu23 72 72 AG 16 222C 23042A 23098D 23076A 23083A 49 10 P. syringae PNA29.1a 20795B 23094C 23074A 100 BE60 23095C 100 Curtobacterium sp. S121 C. flaccumfaciens 11.1 23046B 23048C 100 M. yunnanensis L7-617 100 23085B Paenibacillus sp. 9N4 100 AG 16 2268C D E. aurantiacum BVC2 100 149 22 Staphylococcus sp. SV3 AG 16 2268A D AG 16 2268B D 99 B. simplex CCMM B622 95 23079A 23097A 99 23013A B. licheniformis T10 93 AG 16 1909C 98 B. atrophaeus BPRIST036 B. subtilis SJ01 98 126 8 23052A 94 23023C 23044A

Erwinia

Pantoea

Serratia Rahnella

Pseudomonas

Curtobacterium

Micrococcus Paenibacillus Exiguobacterium Staphylococcus

Bacillus

20

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Know yourself – What's our bet line of defence?

Detection  Molecular tests – more time efficient, sensitive and are culture independent  Evaluation of E. amylovora specific PCR -

95 strains of bacteria, 8 common PCRs 7 out of 8 tests tested gave unsatisfactory results Both false negatives and false positives A number of the false positives were with bacteria isolated from our surveys

Powney et al (2011) The specificity of PCR-based protocols for detection of Erwinia amylovora. Australas Plant Pathol 40(1): 87-97

 One primer set robust in this evaluation  No diagnostic is perfect so reliance on one marker is not the best practice solution – like with other important pathogenic bacteria a multiplex approach is more desirable. biosecurity built on science


Know your enemy  We know the host range  We know there are host specific-groupings

Spiraeoideae-infecting strains (Apple strains)

- Spiraeoideae-infecting - Rubus-infecting

 Do we know what causes host specificity?  Do we know how much genetic diversity there is between strains?

Rubus-infecting strains

Need to be considered to in diagnostic design

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Know your enemy – Genetic diversity of E. amylovora

The Pan-genome of E. amylovora  12 Genomes of E. amylovora (we sequenced six for this project)

- 9 Spiraeoideae-infecting (very little genetic diversity) - 3 Rubus-infecting (more genetic diversity)

Powney et al (2011) Genome sequence of an Erwinia amylovora strain with pathogenicity restricted to Rubus plants. J Bacteriol 193(3): 785-786

  

5733 genes 3414 core gene shared by all strains Average E. amylovora genome has Phylogenetic tree based on core genome 3819 genes: 90% of each individual genome is core -> good for diagnostics Compared to similar types of analysis with other plant pathogenic bacteria E. amylovora is not a genetically diverse species biosecurity built on science


Know your enemy – Do we know what causes host specificity?

The Pan-genome of E. amylovora Plasmids

LPS

Rezzonico et al (2012) Lipopolysaccharide biosynthesis genes discriminate between Rubus- and Spiraeoideaeinfective genotypes of Erwinia amylovora. Mol Plant Pathol

Hrp Pathogenicity island

T6SS variation

Mann et al (2012) Comparative analysis of the Hrp pathogenicity island of Rubus- and Spiraeoideaeinfecting Erwinia amylovora strains identifies the IT region as a remnant of an integrative conjugative element. Gene.

Putative secondary metabolite cluster (PKS &NRPS) Mann et al (in preparation) The pan-genome of E. amylovora

Arabinose biosynthesis cluster biosecurity built on science


Know your enemy – using the knowledge to better our defences

Diagnostic design –drawing on our knowledge Unique target identification pipeline

Ea core genome

vs

Function

E. tasmaniensis

Erwinia Ejp

Location

E. pyrifoliae

Ea-potential targets

NCBI database

Ea-specific targets

HGT

E. billingiae

Analyse for suitability as a diagnostic target

Primer design

66 regions of sequence collectively spanning 101561 bps Powney et al (2011) An analysis of the Erwinia amylovora pan-genome identifies novel chromosomal targets for molecular diagnostics Acta Hort 896

E. amylovora specific multiplex-PCR

Rubus-infecting E. amylovora specific multiplex-PCR

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Thank you  For more information, please email rachel.mann@dpi.vic.gov.au   

Dr Brendan Rodoni Dr Kim Plummer Dr Jo Luck

    

Prof Steven Beer Dr Tim Sawbridge Dr Brion Duffy Dr Theo Smits Jean Bonasera

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Phylogenetic tree based on SNP variation in the Spiraeoideae-infecting strains

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% core

Pan-genome

Species

CDS

No. Genomes

type

Escherichia coli

44%

17

Open

Ralstonia solanacearum

48%

6

Unknown

Xanthomonas oryzae

63%

4

Open

Pseudomonas syringae

64%

19

Unknown

Streptococcus pneumoniae

74%

44

Open

Xanthomonas campestris

75%

5

Open

Listeria monocytogenes

80%

26

Open

Staphylococcus aureus

85-89%

17

Closed

Erwinia amylovora

90%

12

Open

Mycobacterium tuberculosis

98%

9

Closed

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ICE in the hprPAI of Erwinia sp

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Erwinia ancestor

E. billingiae

Pathoadaptation - Acquisition of Hrp T3SS cluster - Acquisition of ICE

Pathoadapted Erwinia ancestor

Loss of ICE due to genome rearrangement

E. tasmaniensis

- Deletion of AttL and related genes - Acquisition of clpB and genes encoding EF-hand domain proteins

Large-scale adaptations

E. piriflorinigrans

E. pyrifoliae

E. amylovora

In each strain individually: - Deletion of smaller regions in ICE - Modification of the EF-hand domain region

Adaptation due to reductive genomics Spiraeoideaeinfecting isolates

Rubus-infecting isolates

Evolution of hrpPAI region in pathoadapted Erwinia sp. biosecurity built on science


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