Lyme disease

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Lyme Disease Dr. Peter Dobie


Introduction 

Lyme disease was named in 1977 when arthritis was observed in a cluster of children in and around Lyme, CN

Conditions suggested that this was an infectious disease probably transmitted by an arthropod

Further investigation revealed that Lyme disease is caused by the bacterium


Causative Organism 

Borrelia burgdorferi

Loosely coiled spirochete

8-20 micrometers


Ticks that cause Lyme disease

Lone Star Tick

Black-legged Tick

Rocky Mountain Tick


Three Stages of Disease 

Localized rash – erythema chronicum migrans

Dissemination to multiple organ systems

Chronic disseminated stage often with arthritic symptoms


Localized Rash


Dissemination 

Signs of early disseminated infection usually occur days to weeks after the appearance of a solitary erythema migrans lesion

Neurologic – Bell’s Palsy

Musculoskeletal manifestations migratory joint and muscle pains

Late disseminated Lyme disease is intermittent swelling and pain of one or a few joints.

may

include


Chronic Disseminated 

Chronic arthritis

Chronic axonal polyneuropathy

Lyme disease morbidity may be severe, chronic, and disabling.

Rarely, if ever, fatal


Diagnosis ď‚—

Diagnosed clinically, confirmed serologically.

ď‚—

Often appropriate to treat patients with early disease solely on the basis of objective signs and a known exposure.

ď‚—

CDC recommends testing initially with a sensitive first test, ELISA or an IFA test, followed by testing with the more specific Western immunoblot (WB) test to corroborate equivocal or positive results obtained with the first test.


Serology 

Patients with early disseminated or late-stage disease usually have strong serological reactivity

Antibodies often persist for months or years following successfully treated or untreated infection.

Seroreactivity alone cannot be used as a marker of active disease


Problems with Serology 

IFA false positive may occur if patient has syphilis, relapsing fever or RA.

IFA interpretation highly subjective

EIA lacks sensitivity in early disease.

EIA false positives with syphilis, other treponemes, IM and autoimmune disease.


Western Blot 

Must be used if the Lyme IgG/IgM antibody serology is equivocal or positive

"Osp" refers to outer surface protein of the bacteria.

"kDa" is the abbreviation for "kilodalton," which is used for molecular weight designations.

Lyme antibodies of importance are against the following molecular weights of the B. burgdorferi antigens: 23-25 kDa (Osp C); 31 kDa (Osp A); 34 kDa (Osp B); 39 kDa; 41 kDa; and 83-93 kDa7.


Treatment 

Single dose doxycycline shortly after tick bite.

Lyme disease give doxycycline followed by amoxacillin

Neuroborreliosis requires IV antibiotic therapy.


Address: SUITE 3A, EDGECLIFF COURT, 2 NEW McLean ST, EDGECLIFF, NSW 2027 Email: info@drpeterdobie.com Telephone: 02 9362 0493 Fax: 02 9363 0767

Dr. Peter Dobie

Thank You


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