Anandamide - 94421-68-8

ANANDAMIDE (AEA) (94421-68-8) Anandamide, also known as N-arachidonoylethanolamine or AEA, is a fatty acid neurotransmitter derived from the non-oxidative metabolism of eicosatetraenoic acid (arachidonic acid) an essential ω-6 polyunsaturated fatty acid.

Status:

In Mass Production

Unit:

25kg/Drum

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1. Anandamide (AEA) Specifications

Name:

Anandamide (AEA)

CAS:

94421-68-8

Purity

50% powder；85% oil

Molecular Formula: C22H37NO2 Molecular Weight:

361.526 g/mol

Melt Point:

−4.8°C

Chemical name:

N-Arachidonoyl-2-hydroxyethylamide

Synonyms:

Anandamide; AEA; Arachidonylethanolamide; Arachidonoyl ethanolamide.

InChI Key:

LIGWYLOEDYSMTP-DOFZRALJSA-N

Half Life:

N/A

Solubility:

Soluble in DMSO, Methanol, Water

Storage Condition:

0 – 4 C for short term (days to weeks), or -20 C for long term (months)

Since Anandamide (AEA) synthesizes in areas of the brain where memory, motivation, cognitive processes and movement control are managed, it can

Application:

influence physiological systems such as pain, appetite regulation, pleasure and reward.

Appearance:

light yellow powder

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2. What is Anandamide (AEA) CAS 94421-68-8?

Anandamide, also known as N-arachidonoylethanolamine or AEA, is a fatty What is Anandamide (AEA) CAS 94421-68-8?

Anandamide, also known as N-arachidonoylethanolamine or AEA, is a fatty acid neurotransmitter derived from the non-oxidative metabolism of eicosatetraenoic acid (arachidonic acid) an essential ω-6 polyunsaturated fatty acid.

The name is taken from the Sanskrit word ananda, which means “joy, bliss, delight�, and amide. It is synthesized from N-arachidonoyl phosphatidylethanolamine by

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multiple pathways. It is degraded primarily by the fatty acid amide hydrolase (FAAH) enzyme, which converts anandamid.

3. Anandamide (AEA) CAS 94421-68-8 benefits Anandamide (AEA) is synthesized enzymatically in the areas of the brain that are important in memory, thought processes and control of movement. Research suggests that anandamide plays a role in the making and breaking of short-term connections between nerve cells, and this is related to learning and memory. Animal studies suggest that too much anandamide induces forgetfulness. This suggests that if substances could be developed that keep anandamide from binding to its receptor, these might be used to treat memory loss or even to enhance existing memory!

4. Anandamide (AEA) CAS 94421-68-8 Mechanism Of Action?

Anandamide, also known as N-arachidonoylethanolamine (AEA), is a fatty acid neurotransmitter derived from the non-oxidative metabolism of eicosatetraenoic acid (arachidonic acid), an essential omega-6 fatty acid. The name is taken from the Sanskrit word ananda, which means “joy, bliss, delight�, and amide. It is synthesized from N-arachidonoyl phosphatidylethanolamine by multiple pathways.

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5. Anandamide (AEA) CAS 94421-68-8 Application Anandamide (AEA) synthesizes in areas of the brain where memory, motivation, superior cognitive processes and movement control are managed. In this way, it influences physiological systems such as pain, appetite regulation, pleasure and reward.

6. Anandamide (AEA) powder for sale(Where to Buy Anandamide (AEA) powder in bulk)

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Our company enjoys long term relationships with our clients because we focus on customer service and providing great products. If you are interested in our product, we are flexible with the customization of orders to suit your specific need and our quick lead time on orders guarantees you’ll have great tasting our product on-time. We also focus on value-added services. We are available for service questions and information to support your business.

We are an professional Anandamide (AEA) powder supplier for several years, we supply products with competitive price, and our product is of the highest quality and undergoes strict, independent testing to ensure that it is safe for consumption around the world.

References Cui HJ, Liu S, Yang R, Fu GH, Lu Y. N-stearoyltyrosine protects primary cortical neurons against oxygen-glucose deprivation-induced apoptosis through inhibiting anandamide inactivation system. Neurosci Res. 2017 May 9. pii: S0168-0102(17)30012-3. doi: 10.1016/j.neures.2017.04.019. [Epub ahead of print] PubMed PMID: 28499834.

King-Himmelreich TS, Möser CV, Wolters MC, Schmetzer J, Schreiber Y, Ferreirós N, Russe OQ, Geisslinger G, Niederberger E. AMPK contributes to aerobic exercise-induced

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antinociception downstream of endocannabinoids. Neuropharmacology.

2017 May 4. pii: S0028-3908(17)30198-3. doi: 10.1016/j.neuropharm.2017.05.002.

[Epub ahead of print] PubMed PMID: 28479394.

Pirone A, Lenzi C, Briganti A, Abbate F, Levanti M, Abramo F, Miragliotta V. Spatial distribution of cannabinoid receptor 1 and fatty acid amide hydrolase in the cat ovary and oviduct. Acta Histochem. 2017 May;119(4):417-422. doi: 10.1016/j.acthis.2017.04.007.

Epub 2017 May 4. PubMed PMID: 28478955.

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