Steve Ingham - Recovery and adaptation

Page 1

Recovery and adaptation Dr Steve Ingham Head of Physiology, English Institute of Sport, UK


250 staff

Centres at – – – – – – – –

Loughborough University Sportscity, Manchester, Sheffield, Gateshead, Bisham Abbey, Bath University, Alexander Stadium/Lilleshall, London (Twickenham, Brunel University, Lee Valley) – Milton Keynes

– – – – – – – – –

Medicine Physiotherapy Strength & Conditioning Physiology Performance Analysis Biomechanics Nutrition Psychology Performance Lifestyle Performance Lifestyle

4000 hours of support per week

88% 83% 95%


91%

84% 84%



“Are y you going g g to make me go faster?”


“Just Just tell me if I am talking crap”

“D ’t ruin “Don’t i th the hi high-jump!” hj !”



Training categorisation Training categorisation 9 8 7 6 5 4 3 2 1 0

VO2

Heart Rate 200

LT2

VO2 max

160

Race Pace

120 80

He eart rate (b b/min)

Blood lactate (mM) Oxyygen uptakke (l/min)

Blood Lactate

40 0 14

15

16

17

18

19

20

21

22

23

Running speed (km/hr)

P Proportio n of total training vvolume (% %)

90 80 70 60 50 40 30 20 10 0

Sub‐LT2 Sub LT2

LT2‐VO2max LT2 VO2max

Cycling

70

20

10

Swimming Canoeing

77 60

16.4 30

6.6 10

Athletics hl

50.7

29.6

19.7

Training category

VO2max‐Race VO2max Race Pace + Pace +


What are the ‘stresses’ in running? What are the stresses in running?

*my notes only

Comparison between sports of rowing and athletics

Injury

82

3

Over‐training'

15

4 73 2 0


15-17

Forces through single limb on Forces through single limb on ground contact x body mass

7-9

3-6 3-5 2-4

Source: Dr Paul Brice, Biomechanist, English Institute of Sport


Eccentric load •

Concentric (contraction) and eccentric (lengthening ( g g action) ->Eccentric phase involves fewer muscle fib fibres ffor a given i fforce ->Strain on contractile and structural components of the muscle ->Micro-trauma

The issues in athletics/running The issues in athletics/running • Key observations – ‘Over‐training’ cases are rare g – Problems with injury/tolerance of mechanical loading (soreness) are high loading (soreness) are high – Use of recovery treatments is high – 16/27 are ‘attached’ to recovery treatment / ‘ h d’ – Role of other support disciplines!


Thoughts on adaptation Thoughts on adaptation Some distinctions to be made 1. Training response vs injury 1 2. Maximum adaptation vs maximum t i i lload d training 3. Training vs competition

How do you get fit? How do you get fit? Mechanical (inc neural)/ metabolic stimulus

Train

 Hormonal,, immune response  inflammation  breakdown  gene transcription  growth

Blood flow/ Eat/Drink Nutrients

Adapt p

No further stress t

Rest


Jean Baptiste Lamark Jean Baptiste First Law: Fi L I every animal In i l which hi h has h not passed d the h limit li i of its development, a more frequent and continuous use of any organ gradually strengthens, strengthens develops and enlarges that organ, and gives it a power proportional to the length of time it has been so used; while the permanent disuse of any organ imperceptibly weakens and deteriorates it, and progressively diminishes its f ti functional l capacity, it until til it finally fi ll disappears“. di “

-v ve

Performance

+ve +

Stress/Adaptation

Time (hours, days)


R Repeated bout effect t d b t ff t

-ve

Performanc P ce

+ve

» Presenting the same stimulus to the body again » Reduced disturbance in homeostasis » BUT will probably result in smaller adaptive response

Time (hours, (hours days)

vs vs.

trained Gene Ex xpression

Gene Ex xpression

untrained

Flueck M. (2010) Myocellular limitations of human performance and their modification through genome‐dependent responses at altitude. Exp Physiol. 95(3):451‐62


Diminishing returns Diminishing returns

Perry et al (2010). Repeated transient mRNA bursts precede increases in transcriptional and mitochondrial proteins during training in human skeletal muscle. J Physiol. 1;588 4795‐810

Strength changes vs stimulus Strength changes vs stimulus 100

*

Performan nce

MVC (N.m)

+ve

90 80 70 60 WARM CON

-ve

50 40

Pre Day 1 Day 2 Day 3 Day 4 Day 7

Time (hours, days)

Time

Ingham et al (2010) Effect of a concentric warm-up exercise on eccentrically induced soreness and loss of function of the elbow flexor muscles. J Sports Sci. 2010 Nov;28(13):1377-82.


Recovery treatments Recovery treatments

Progress? 2002‐5

2012


Perceivved sorenesss (1‐10) Isomettric force (Nm.kg‐1)

C t l Control

CWI

0

24

Ice baths Ice baths

8 6 4 2 0 Pre

1

48

168

35 3.5 3 2.5 2 1.5 1 Pre

24 48 Time (hours)

168

10-15° 10 15° C water 10-15 mins

Bailey et al, 2007 Influence of cold-water immersion on indices of muscle damage following prolonged intermittent shuttle running. J Sports Sci.Sep;25(11):1163-70.

Magnetic Resonance Imaging p –p plantar flexion 12 sets of 5 repetitions T2 relaxation time as marker of intra muscular water Control

Cold water immersion 15 mins at 5 deg

Yanagisawa et al (2003) Evaluations of cooling exercised muscle with MR imaging and 31P MR spectroscopy. Med Sci Sports Exerc. 2003 Sep;35(9):1517-23.


Meta analysis of CWI Meta‐analysis of CWI

CWI has a moderate effect in alleviating soreness from exercise in general

CWI;

CWI is highly effective in alleviating soreness after high intensity exercise

CK efflux CK CK efflux ‐ efflux ‐ reduced Muscle strength –– little effect Muscle strength Muscle power –– helps recovery Muscle power

Soreness

Leeder et al (2011) Cold water immersion and recovery from strenuous exercise: a meta-analysis. Br J Sports Med. 22

“Ice-baths help me to run the day after a hard session” Do workout Recovery therapy h must work

Feel sore

Unable to complete next session

Able to complete next workout

Feel less sore

Do next workout Use recovery therapy


Long term adaptation? g p Pe ercentage e change (%)

16 14

Cooled

Control

12 10

8 6 4 2 0 -2 -4 Perfomance trial

VO2max

VT

Test parameter

Femoral artery diameter

Yamane et al., 2006 Post-exercise leg and forearm flexor muscle cooling in humans attenuates endurance and resistance training effects on muscle performance and on circulatory adaptation. Eur J Appl Physiol. 2006 Mar;96(5):572-80. Epub 2005 Dec 22.

Ibuprofen Maximal over‐the‐counter d dose • (IBU 1200mg.day, ACET (4000mg day) (4000mg.day) • Double‐blind placebo controlled • 10‐14 sets of 10 eccentric reps at 120% of concentric reps at 120% of concentric max of knee extensors

Measured skeletal muscle protein synthesis 24hours i h i 24h after damaging exercise via Fractional Synthesis Rate Synthesis Rate ‘Fractional (FSR).

Blocking COX‐2 pathway, reduces inflammation and soreness. Trappe et al.,m (2002) Effect of ibuprofen and acetaminophen on postexercise muscle protein synthesis. Am J Physiol Endocrinol Metab. 2002 Mar;282(3):E551-6.


Repeated sprint performance pre-post recovery and 3 week gain

10

Control Skinz Cold D25mg D50mg

24h post training relative to pre-training

8

6

b a a a

4

a

a

a

% gain on repeat sprints (mean) a

a

a

2

0

2 -2

-4

-6

Week 1 Week 2 a: p<0.05; b: p<0.1

% gains from 3 Week training block

Week 3

At the end of a short training block cold and D50mg lessened gains

By week 2 only cold and D50mg (x2 p.d p.d)) were Having significant recovery effects Data courtesy of Christian Cook

“Am I just doing more training to get the same effect”

“Ice-baths help me to run the day after a hard session”

Do workout specific to individual needs

Do workout Recovery therapy h must work

Feel sore

Unable to complete next session

Able to complete next workout

Feel less sore

Do next workout Use recovery therapy

Feel less sore

Do next workout

Adjust expectations for training for training following hard sessions

Feel sore

Unable to complet next session

Adjust next session


Periodising recovery treatments Adaptation more important, recovery less important –i.e. mid season/national champs i id / ti l h • • • • • • • •

Active recovery Contrast bathingg Deep water exercise Compression garments Ice baths NSAID Massage Stretching

   × × × -

Yes Yes Yes No (may impair adaptation) No (may impair adaptation) No (may impair adaptation) For other reasons For other reasons For other reasons

Recovery more important, adaptation less Recovery more important adaptation less important –i.e. World Champs, later races • • • • • • • •

Active recovery Active recovery Contrast bathing Deep water exercise Compression garments Ice baths NSAID Massage Stretching

     × -

Yes es Yes Yes Yes Yes No unless medical No unless medical For other reasons For other reasons


Recovery Plan Finish race

What?

Why

After your transition through media and within 5 mins i

Recovery drink, e.g. REGO 100g with 750 ml of water

Rapid replenishment of muscle fuel stores Enhanced muscle recovery and Enhanced muscle recovery and repair Enhanced hormonal profile and immune function

Sort yyour self out then go g straight for a …

Active recovery walk of 10 y mins

Improved metabolic clearance Enhanced immune response Improved readiness for the next session

OPTIONAL depending upon OPTIONAL depending upon your therapy plan (needs to be consistent with previous competitions)

Massage or treatment Massage or treatment

Depends upon plan Depends upon plan

Then either

Contrast bathing Contrast bathing/showers; 15 mins (3 mins warm; 1 min cold x 5) finish on cold,

Improved metabolic clearance Reduced inflammatory response

OR

Ice bath, 10 mins at 10°C water

Reduced muscle damage Reduced soreness

Before you leave the warm‐ up track…

Compression socks on

Improved blood flow preventing pooling in lower limbs pooling in lower limbs Reduced soreness and damage

If you have time when you get back to your room…

Elevate legs for 20 to 30 mins

Reduced blood pooling Reduced inflammation

Percenttage change (%)

Is it better to warm down or sit down? 120

Blo ood lactatte (% of m max)

100

16 14 12 10 8 6 4 2 0 -2 -4

Control Cooled

Perfomance ti l trial

80

VO2max

VT

Test parameter

Passive

60

Massage Run

40

Bikee 20 0 5

10

15 20 Time post (min)

25

30

Femoral artery t diameter


PGC1 1 α mRNA

8

Pre

30 0 s

VEGF mR RNA / RP2 (A A.U.)

vs

4 mins

120s

3 fold

4 mins

30 0 s

4 mins

30 0 s

30 0 s

Post‐exercise response to interval or all‐out p

Post

6 4 2 0

5

Pre

Post

4 3 2 1 0

Int

Cont

Int

Cont

Gibala, et al (2009). Brief intense interval exercise activates AMPK and p38 MAPK signaling and increases the expression of PGC‐1a in human skeletal muscle. J. Appl. Physiol. 106: 929–934. Taylor et al., (current study ). Interval and all‐out continuous exercise activates PGC1 a and VEGF signaling in trained athletes

Maximum adaptation is the focus not maximum training Physiology of Performance Potential for energy turnover

Minimising energy expenditure in performance Tactical method

Preparedness

Economy

Physical Pacing Fast-start Even pace

Energy expend Drafting Minimal distance covered

Anthropometric

Ergogenic

Optimal body mass Body comp Fat oxidation Carnitine

CHO drink Bicarb/citrate Caffeine PostCHO/BCAA di k drink

High resistance reps High Intensity warm-up Post – 15 min cool down Ice bath

Anaerobic

Aerobic

Lactate Threshold Th h ld Max lactate steady state reps

Mileage Hill reps Mech/S&C Glut/Ham strength Drills Core stability Compression stockings Fat oxidation

VO2max Peripheral uptake Volume/wk CHO intake Central delivery vVo2max V 2 reps Lung fitness Altitude Hb Ferritin stores Blood volume Tissue hydration Immune function

Anaerobic Capacity

Power

20-90s reps 1-2 sessions/wk CHO intake/ protein Bi-carb β-alanine

Plyometrics Explosive starts Sprint mechanics Caffeine Creatine Resistance training Hormonal response BCAA


Athl t P Athlete Profile fil – June J 2002 Mean ± SD Range

June 2002

Economy (ml.kg-1.km.hr-1)

226

LT (km.hr-1)

16

VO2max (ml.kg-1.min-1)

70

speed VO2max (km.hr-1)

19.4

• International International 5 5 years • Commonwealth Commonwealth champion g • Plateauing in physiology and performance • Coach questions focussed on use of recovery f treatments

Training intensity profile: g yp Male 1500m runner Proportion o of total traaining volume (%)

60

Pre Post

50 40 30 20 10 0 <80

80 to 90

90 to 100 100 to 110 110 to 120 120 to 130 % of VO2max

Ingham, S.A., Fudge, B.W. and Pringle, J.S. (2012) The Change In Training Distribution, Physiological Profile And Performance For A Male International 1500m Runner. IJSPP. 7(2):193-5.


15 Perce entage change (% %)

Year 1

Year 2

10

5

0

-5 5

VO2max

vLT

vVO2 max

RE

The change (%) in physiological measures for year 1 and year 2 (mean of Nov, Mar, Sept) compared with Sept T0 and Sept T1, respectively. NB RE, negative change = RE improvement ).

Yr2

Pre-support

Yr 1

Ingham, S.A., Fudge, B.W. and Pringle, J.S. (2012) The Change In Training Distribution, Physiological Profile And Performance For A Male International 1500m Runner. IJSPP. 7(2):193-5.

Figure 3. The change in 1500m performance speed during competitive races prior to physiological support and during year 1 and 2. Ingham, S.A., Fudge, B.W. and Pringle, J.S. (2012) The Change In Training Distribution, Physiological Profile And Performance For A Male International 1500m Runner. IJSPP. 7(2):193-5.


Training intensity profile: g yp Male 1500m runner Proportion o of total traaining volume (%)

60

Pre Post

50 40 30 20 10 0 <80

80 to 90

90 to 100 100 to 110 110 to 120 120 to 130 % of VO2max

Ingham, S.A., Fudge, B.W. and Pringle, J.S. (2012) The Change In Training Distribution, Physiological Profile And Performance For A Male International 1500m Runner. IJSPP. 7(2):193-5.

Overview Overview • Running is different to most other linear energetic sports due to the neuro‐muscular pattern • Injury rate is high, probably due to high eccentric loading • Recovery treatments are popular • Recovery treatments are not encouraged out of Recovery treatments are not encouraged out of competition (this has taken 8 years to change) • Recovery treatments are used in competition Recovery treatments are used in competition • Focus is returning to understanding and intervening with training first before recovery treatment is considered training first before recovery treatment is considered


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Nigel Walker Ken van Someren


Ferguson occlusion g

800

800

600

600

400

400

200

200

0

0 Bout 1 Bout 2 Bout 3 Bout 4

0‐30

30‐60

60‐90 90‐120


Male

25

Female

Proportion of tottal training volum me ((%)

30

20 15 10 5 0 LT LTP Training intensity

Go old med dals won

20

vVO2max

Physiologists at recent Olympics l i

19 11

Golds

15

Linear (Golds) 11 10

8

9

y = 2.56x y 2.56x ‐ 9.2 R² = 0.999

7

5 1

4

Top the medal table = 24 All 302 golds = 122 0 golds = 3.5

0

3

4

5 6 7 8 9 10 11 12 No. of Physiologists at Olympics

I f Infra‐structure and investment di


Training intensity

How do your athletes actually train?

Time (a microcycle)

• Poor association between intensity prescribed and performed (r=0 and performed (r 0.2 2‐0 0.6) 6)

Stewart AM, Hopkins WG. (1997) Swimmers' compliance with training prescription. Med Sci Sports Exerc. 29(10):1389-92. Hewson DJ, Hopkins WG (1996) Specificity of training and its relation to the performance of distance runners. Int J Sports Med. 17(3):199-204.


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