Medical Forum March 2020 - Public Edition

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CLINICAL UPDATE

Key messages

Chronic craniofacial pain can be difficult to manage

Current treatments often have adverse effects

Non-destructive treatments offer analgesia and are simple to apply. radiofrequency treatment. Occipital neuralgia can respond to radiofrequency thermal lesions of the second cervical dorsal root

ganglion and third occipital nerve with quite prolonged periods of remission but, again, at the price of temporary post cervical area altered sensation and ‘burning’ dysaesthesias. Non-destructive electrical stimulation of cranial nerves or upper cervical nerve roots (neuromodulation) can provide very good analgesia with reduced downside risk as these electrodes are implanted subcutaneously through an introducing needle under ultrasound control.

The active electrode segments are placed adjacent to craniofacial sensory nerves including the supra and infraorbital nerves, the greater and lesser occipital nerves and even uncommon targets such as the auriculotemporal nerve and mandibular division nerves. An alternative placement in the cervical epidural space increases risk but can be effective for craniofacial pain states. Implanted systems can always be removed with little loss for the patient except perhaps financial expense, as these systems can be pricey. The introduction of sub-threshold stimulation waveforms in the past decade, especially 10 kHz high frequency stimulation and burst stimulation algorithms, have eliminated the often unpleasant paraesthesia sensations experienced in the face or occiput with earlier low (1-100 Hz) frequency systems. The newer waveforms have been shown to greatly improve the efficacy of stimulation compared with older ‘tonic’ waveform devices.

Sagital and Anteroposterior X-ray images of implanted supra and infraorbital electrodes for high frequency stimulation

Author competing interests – nil

FIVE MINUTE CLINICAL UPDATE Frozen Shoulder (Adhesive Capsulitis) Frozen shoulder is a common condition that causes a great deal of confusion, both from a diagnostic and treatment perspective. Primary or idiopathic frozen shoulder comes on spontaneously, although it often presents with a history of minor injury. There is a strong association with endocrine disorders, particularly diabetes. Women outnumber men. The typical patient is a middle-aged woman who was initially diagnosed with impingement and had some bursitis on her ultrasound. She has had lots of physiotherapy, two subacromial injections with no real response, and is now struggling with sleep and in severe pain. Pain levels are usually high. However, the diagnostic algorithm for frozen shoulder is simple. There are only

two criteria. Firstly, there is a global loss of active and passive range of motion, and x-rays are normal. What does this mean clinically? Understanding the difference between active and passive movement is key. In frozen shoulder, not only can the patient not move the joint fully in all directions (active), but neither can the examiner (passive). External rotation is the first movement usually involved. The only common differential diagnosis is arthritis, which will show up on an x-ray. All other conditions, such as impingement and rotator cuff tearing, will reduce active movement but the passive movement should be preserved. There are only three treatments for frozen shoulder with good clinical evidence to support them. The first, and the mainstay

By Dr David Colvin

of treatment, is glenohumeral cortisone injections. The injections have to be performed into the shoulder joint itself, not the bursa, and this is most commonly done with CT guidance. Within two weeks, there is often a substantial improvement in pain levels, but movement is slower to recover. It usually takes a course of 2-3 injections to see significant results. The other treatments are manipulation under anaesthetic or surgery to release the capsule. In my practice, surgery is almost exclusively reserved for diabetics who experience a more severe frozen shoulder. Frozen shoulder is a condition where symptoms evolve during the treatment phase. It almost always looks like impingement, initially. By the time the patient sees an orthopaedic surgeon, the condition has declared itself, and it’s easy to look like the clever specialist.

Western Orthopaedic Clinic is a multi-centre orthopaedic group, with rooms across Perth. We are committed to bringing the newest techniques and are involved in research in many areas.

Phone: +61 8 9489 8700 Email: woc@wocwa.com wocwa.com

MEDICAL FORUM | PAIN MANAGEMENT ISSUE

MARCH 2020 | 39


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Social Pulse Christmas: SGJ Midland Hospital, SJG Murdoch, SJG Subiaco, Ramsay, Bethesda Health Care; SJG Mt Lawley

9min
pages 56-62

Wine Review: Sittella Dr Martin Buck

7min
pages 53-55

Knee AO

4min
pages 51-52

US Prostate Testing

2min
page 50

Breast Implant Update

5min
pages 46-48

Diabetes Testing

3min
page 49

AI in Medicine

5min
pages 44-45

Women’s Care

2min
page 43

Research Support

4min
pages 41-42

AI in Radiology

3min
pages 39-40

Stereotactic Body Radiotherapy

2min
page 38

Real World Research

2min
page 37

Clinician-led technology

12min
pages 20-23

Vaping

4min
pages 30-31

Superannuation – Rob Pyne

5min
pages 34-36

Australian National Phenome Centre

11min
pages 24-27

Silicosis

6min
pages 28-29

WA Digital Health Strategy

5min
pages 18-19

Genetic Testing in Cancer

13min
pages 7-9

WA News

4min
page 12

Research Briefs

4min
pages 16-17

Global News

3min
pages 14-15

Opinion: Overdiagnosis – Dr Joe Kosterich

2min
page 6

Local Brief

4min
page 13

Q&A: Dr Andrew Miller

8min
pages 10-11
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