C19 - SAGER - teaching rheumatology

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a teaching method for rheumatology Simplifying rheumatologic diagnosis at the bedside AFLAR May 2011 Daniel Sager MD, FACR


introduction Rheumatology is often perceived by students to be a complex subject, and difficult to learn 1  Diseases may have:  diverse and overlapping manifestations  features similar to infection, malignancy, toxicities, or even psychosomatic disorders  serious consequences 


introduction (2) 

Rheumatic complaints and diseases are a common part of medical practice2  

>15% of visits to primary care physicians 6-10% of population may have chronic rheumatic disability

yet some rheumatic diseases are rare


introduction (3) Rheumatology is generally under-represented in medical education3  Novice learners use their limited experience in generating only one or two diagnostic hypotheses, often by recall of a past image  Diagnostic reasoning, and diagnostic success, is limited if the differential diagnosis is incomplete 


introduction (4) Although comprehensive content guidelines for rheumatologic diagnosis are available4, these do not include orientation of the student to the primary categories, or patterns, of disease  Clinical settings allow for little teaching time 


therefore 

A simplified classification system, to teach and to use at the bedside in the evaluation of patients with rheumatic disease, is needed


teaching rheumatologic diagnosis First, teach a simplified system to organize diagnostic possibilities (a menu)  Second, teach the clinical disease patterns  Third, teach diagnostic reasoning (deductive testing of hypotheses)5 


applying a logical sequence


the rheumatologic menu FIVE CATEGORIES

CTD Vasculitis Arthritis Non-articular pain Miscellan. 1 2 3 4 5


interviewing with a rheumatology menu Key to success usually involves simplification of complexity  Classification schemes organize complex data  “Layering” of knowledge, at a later stage, can be customized to the particular student and teaching environment using any effective teaching technique6,7 


Carl Linnaeus (1707-1778) father of modern taxonomy


diagnosis is the central element in patient care Success of treatment is dependent on accuracy of diagnosis  Patient compliance with treatment is based on agreement with physician’s diagnosis 


The rheumatologic menu CTD

Vasculitis

ARTHRITIS Non-articular pain

Misc.


The rheumatologic menu CTD

Vasculitis

Arthritis

Non-articular pain

Misc.


connective tissue disease (CTD) 3 groups, each with 2 options SLE/Sjogren syndrome  Polymyositis/Dermatomyositis  Systemic sclerosis (scleroderma) 

 Diffuse

and rapidly progressive form  Limited form (CREST), or localized form Overlap syndromes and differential diagnosis


SLE 

AFTER recognizing the diagnostic pattern, look for ways to make the diagnosis complete (if needed)  Diagnostic

criteria

 Serology  Biopsy  Rule

out drug induced lupus  Rule out mimics  Expert opinion


The rheumatologic menu CTD

VASCULITIS

Arthritis

Other MS pain

Miscellan.


vasculitis 3 groups, defined by size of vessel involved

Large  Medium  Small 

Medium and small vessel disease features and classification often overlap Beware: many mimics of vasculitis exist, and vasculitis is relatively uncommon


vasculitis: 3 groups, each with 2 options 

Large vessel disease  Giant

cell arteritis (GCA): age > 50 years at onset  Takayasu arteritis: age < 50 

Medium vessel disease  Pulmonary-renal

syndromes (e.g. Wegener’s)  Polyarteritis nodosa 

Small vessel  Specific

diseases  Non-specific (antigenic) causes


small vessel vasculitis 

Specific forms  Henoch

Schonlein Purpura  Mixed cryoglobulinemia: Hepatitis C 

Nonspecific forms  Drug  Tumor  Infection  Autoimmune

(including CTDs and RA)  Other or unknown cause


The rheumatologic menu CTD

Vasculitis

ARTHRITIS vs. Other MS pain

Miscellan.


“joint pain”: arthritis or not?


non-articular pain  

   

Regional pain syndrome (tendon, bursa, muscle) Central pain syndrome (fibromyalgia, complex regional pain) Nerve (peripheral neuropathy, nerve root, cord) Referred (from viscera, spine, other) Internal joint derangement (meniscus, ligament) Bone (fracture, osteonecrosis, tumor, metastasis, other) Skin, fascia, pannus


arthritis or not? 

History: 2 key questions  “Point

to where you have pain”  “What-where-when, otherwise” 

Physical aspects of arthritis Localized pain on passive joint ROM (watching patient’s face) Joint-line tenderness > other tenderness Joint effusion or deformity


2 key questions for pattern recognition Point to where you hurt?  Clarifies if the process is arthritis or not  What else, where else, and when else?  Clarifies if systemic illness features are present or not, or if other disease is present that may underlie the rheumatologic illness or influence treatment of it  Clarifies if process is monoarticular or poly  Clarifies if problem is truly “acute” 


diagnosis is based on pattern recognition 

Diagnosis is formed with the HPI8  HPI

must be more than an inventory of problems  Clinical reasoning skills form the foundation for effective doctoring9 

Diagnosis is tested with the physical exam  Systematically

(adds 5% to case finding)10  Reflexively, to test hypotheses generated11 

Diagnosis is confirmed primarily by excluding alternatives, observing over time, and, only occasionally, with laboratory tests or imaging


steps in “intentional interviewing” Keep diagnostic categories (menu) in mind  Index of suspicion: for common and serious problems  Key questions for pattern recognition  Recognizing the pace of illness  Rheumatologic Review of Systems  Determining the pre-test probability of disease 


pace of illness as clue to diagnosis 

Episodic/progressive?  Lupus  Crystal

arthritis

Acute/emergent?  Infection;

Subacute/additive?  CTD;

Giant cell arteritis; transverse myelitis

Polymyalgia rheumatica/vasculitis; RA

Chronic/progressive?  Osteoarthritis;

fibromyalgia


acute arthritis Patient complains of “joint” pain  Step 1: arthritis or not? 

Step 2: acute or chronic?

Acute arthritis  Infection (bacterial, viral, reactive) or  Crystal associated arthritis and differential dx:  Sarcoidosis (Lofgren syndrome)  Early chronic arthritis (e.g very early RA)  other


chronic arthritis Step 1: arthritis or not?  Step 2: acute or chronic arthritis? 

Step 3: inflammatory arthritis or not?  Chronic

inflammatory arthritis: Rheumatoid arthritis or spondyloarthropathy  Chronic non-inflammatory arthritis: Primary or secondary osteoarthritis, hemarthrosis, hemochromatosis, other


inflammatory arthritis pattern Stiffness syndromes (> pain, weakness)  AM stiffness prominent and prolonged  Worse with inactivity  Signs: heat, swelling, tenderness, redness  Acute phase reactants  Beware: nonspecific or variable features 


Step 4: RA versus Spondyloarthropathy        

Polyarthritis UE predilection Ø back/DIP Extensor tenosynovia Scleritis Nodules, Sjogren’s ANA, RF, CCP Age at onset: any

     

 

Mono/oligoarticular LE predilection + back/DIP joint Enthesitis Uveitis Psoriasis, colitis, urethritis, stomatitis HLA B27 Age at onset: early


the diagnostic pattern of RA S1 (Six weeks)  S2 (Synovitis)  S3 (Several swollen joints)  S4 (Symmetric)  S5 (Small joints) 

Chronic  Inflammatory  Polyarthritis 

Bilateral, in wrists and/or MCP joints


RA pattern and differential dx Severe  Chronic  Inflammatory  Polyarticular  Arthritis  Idiopathic 

Rules out most cases of: celiac disease parvovirus B19 osteoarthritis TB/Lyme fibromyalgia/other crystal arthritis


how are you sure it is RA? 

AFTER identifying the pattern, look for ways to make the diagnosis complete  Diagnostic criteria (90% specificity)  Serology (Anti-CCP antibody: 90% spec.)  Rheumatoid nodule  Radiographs (typical erosions: not early in course, and only in 70% of patients overall)  Rule out mimics  Expert opinion


spondyloarthropathies Ankylosing spondylitis or psoriatic arthritis  Reactive arthritis (+/- inflammatory bowel disease or chlamydia) 

Differential diagnosis may include: sarcoidosis atypical infection atypical RA or CTD chronic stage of gout/pseudogout


The rheumatologic menu CTD

Vasculitis

Arthritis

Other MS pain

MISCELLAN.


miscellaneous disorders associated with rheumatic features (examples)

Fever-mucocutaneous syndromes: e.g. Still’s, Behcet’s, sarcoidosis  Endocrine/metabolic/deposition diseases: thyroid/PTH, acromegaly, diabetes, renal  Malignancy/paraneoplastic syndromes  Hematologic disease: sickle cell, leukemia  HIV syndromes  Drug/toxin exposure  Hereditary/neurologic syndromes 


teaching rheumatologic diagnosis First, teach the menu (schematic) structure  Second, teach the clinical disease patterns  Third, teach diagnostic reasoning 

 Heuristics  Bayes’

Theorem


diagnostic reasoning: examples 

Heuristics (general guiding rules in science)  “What’s common is common” (“Hear hoof beats? Think horses, not zebras”)  Sutton’s law  Uncommon manifestation of common disease > common manifestation of uncommon disease  “Push, shove, or squeeze, give the patient one disease”  Occam’s razor (law of parsimony)  “You have what you have”


using Bayes’ Theorem in diagnosis 

Pre-test probability of diagnosis determines performance of test (post-test probability) i.e. epidemiology and the patient’s disease characteristics (pre-test probability), if calculated accurately by clinician, are critical in interpreting test sensitivity and specificity (and the “result”)


A teaching method for rheumatologic diagnosis Summary  Approach patient with a simplified classification scheme (a menu) in hand  Ask key questions and perform exam to find the predominating pattern  Use bedside diagnostic reasoning to determine pre-test probability of a disease before applying tests


the rheumatologic menu FIVE CATEGORIES

CTD Vasculitis Arthritis Non-articular pain Miscellan. 1 2 3 4 5 SLE/sjog Large PM/DM Medium dSScl/lSScl Small

Acute/chronic Inflamm/non-inflamm RA/spondylo


references 1 

Clawson. It’s past time to reform the musculoskeletal curriculum. Acad Med 2001;76:709 Woolf. Burden of major musculoskeletal conditions. Bull World Health Organ 2003;81:646 Rasker. Rheumatology in general practice. Br J Rheumatol 1995;34:494 Woolf. History and physical exam. Best Prac Res 2003;17:381


references 2 

Elstein. Heuristics and biases: selected errors in clinical reasoning. Acad Med 1999;74:791 Wolpaw. Using SNAPPS to facilitate the expression of clinical reasoning and uncertainties. Acad Med 2009;84:517 Irby. Teaching and learning in ambulatory settings: a thematic review of the literature. Acad Med 1995;70:898 Gruppen. Medical student use of history and exam in diagnostic reasoning. Arth Care Res 1993;6:64


references 3 

Alexander. Moving students beyond an organbased approach when teaching medical interviewing and exam skills. Acad Med 2008; 83:906 Boland. Review of systems, physical examination, and routine tests for case-finding in ambulatory patients. Am J Med Sci 1995;309:194 Benbassat. Suggestions for a shift in teaching clinical skills to medical students: the reflexive clinical examination. Acad Med 2005;80:1121


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